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doi: 10.1073/pnas.2408540121 279 Gheorghita PuscaseluRLobiucADimianMCovasaM

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46 Clinical risk factors for nephrotoxicity secondary to calcineurin inhibitors have included the exposure to the kidney tubules as mediated through transporters, for example, P-glycoprotein, and through drug metabolism enzymes, for example, CYP3A5

It was investigated that the component of BP called linalool holds anti-inflammatory characteristics that are correlated with the suppression of pro-inflammatory variation of mitogen-activated protein kinases (MAPKs) and NF-kB pathways (6)

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Calcitonin Gene-Related Peptide Negatively Regulates Alarmin-Driven Type 2 Innate Lymphoid Cell Responses