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Sharifudin, A
In untreated animals, cleaved caspase-3 (cCasp-3) expression (a marker of cell death induction) is not observed
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lata ) is its resistance against streptomycin and therefore allowing selective growth
Interestingly, another mechanism was proposed as an alternative way to stabilize NRF2 through the interaction between the autophagy adaptor protein p62/sequestosome-1 (p62/SQSTM1) and KEAP1, which leads to the sequestration of KEAP1 in the autophagy-deficient cells and enables NRF2 accumulation [20]
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