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Horrocks, J.L., White, G., and Roberts, L., Journal of Autism and Developmental Disorders , 2008, vol

Miller, P

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Small changes in the time of administration relative to ischemia can shift PRPs effect from anti-apoptotic to apparently pro-inflammatory, as shown in models where PRP given before ischemia produced stronger protective outcomes than when applied at reperfusion 52

The teams findings illuminate how mutations in SLC33A1 disrupt this glutathione transport pathway, offering a molecular explanation for neurodevelopmental disorders such as Huppke-Brindle Syndrome, characterized by severe intellectual disability and progressive neurodegeneration

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