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Similar results were seen in an additional study from this group that identified a modest increase in urinary MEHP in Taiwanese patients with either endometriosis or adenomyosis (Huang et al., 2014)

Disruption of the tight endothelial junction can be achieved via the use of a hyperosmotic agent to produce osmotic disruption or use of a vasoactive compound, bradykinin analogs, or other molecules [3]

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