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Tetraploidization may act as an efficient mechanism to achieve high levels of aneuploidy that increase the biological tolerance of deletion events 38,39 or help reduce the probability of large-scale homozygous losses, which can lead to subpopulations with decreased fitness

To address these issues, most studies focus on the depletion of endogenous GSH by mediators such as nitric oxide (NO) 12 or Cu 2+