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Understanding these mechanisms elucidates the biochemical basis of NO-induced neurotoxicity and its clinical manifestations

For instance, genetic ablation of GPX4 promotes the accumulation of oxidized cardiolipin within mitochondriaa [54], and lipid peroxidation has been reported to emerge initially in mitochondrial membranes before propagating to other cellular compartments

Efficacy of glutathione for the treatment of nonalcoholic fatty liver disease: an open-label, single-arm, multicenter,pilot study

doi: 10.1097/HJH.0000000000001767 114

These data suggest that oral GSH can replete GSH concentrations in several tissues following GSH depletion, such as after toxicologic or pathologic conditions that alter GSH homeostasis

In one study, inadequate zinc intake was observed and zinc deficiency was suggested in patients with MS [27]