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The primary mechanism of action of this peptide relies on the potent stimulation of angiogenesis-a critical process for restoring blood supply in the ischemic fracture site-and the promotion of the survival and proliferation of local cells (e.g., fibroblasts and osteoblasts) [24,29]

Importantly, SELENOP expression was maximal in adjacent astrocytes, rather than the ependymal cells directly lining the ventricles

The synthetic PPARgamma agonist troglitazone inhibits IL-5-induced CD69 upregulation and eosinophil-derived neurotoxin release from eosinophils

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