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But they do it through different molecular mechanisms

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The viability of U87-MG and IR R-1 cells after IR stress and under Rho inhibition is linked to the formation of H2AX foci: IR-resistant clones showed more H2AX foci under stress-free conditions than did U87-MG cells, but this DNA damage sensitivity was rapidly reversed after IR and was still decreased by C3 toxin treatment in parental cells but not in IR R-1 clones (Fig