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Estradiol-17beta and its cytochrome P450- and catechol-O-methyltransferase-derived metabolites stimulate proliferation in uterine artery endothelial cells: role of estrogen receptor-alpha versus estrogen receptor-beta

However, as with any medication, adverse effects can occur, and patients should be informed of potential reactions before commencing treatment

If however significant regression of hyperplasia can occur as an active or passive process, namely by enhanced apoptosis or reduced proliferation, prophylactic surgery could be avoided

4G), raising the possibility of an NRF2-independent mechanism mediated by DMF

I., Stacy, A., Chau, J

A.et al (2007)