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This aberrant activation of PARPs represses the SIRT1 activity and mitochondrial dysfunction, which can be rescued by both PARP inhibitor and NAD + precursors

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Under oxidative stress, CSF-cNs upregulated the activity of ATG4 by combining H 2 O 2 with the cysteine 81 sites of ATG4 and activated ATG4-promoted LC3 lipidation, which is essential for autophagy initiation, and promoted the formation of autophagosomes (Scherz-Shouval et al., 2007

Heskamp S, van Laarhoven HWM, Molkenboer-Kuenen JDM, Franssen GM, Versleijen-Jonkers YMH, Oyen WJG, et al