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Disruptions in the MGBA may play a critical role in both neurodegenerative and neurodevelopmental conditions [56,57,58]

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First, human pharmacokinetic studies must establish how BPC-157 is absorbed, distributed, metabolized, and eliminated in humans oral bioavailability of peptides is typically very low due to enzymatic degradation

Depletion of endogenous GSH sensitizes cells to cuproptosis, whereas GSH supplementation mitigates copper-induced proteotoxic stress 164,168

TCA, tricarboxylic acid cycle