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Homocysteine upregulates hepcidin expression through BMP6/SMAD signaling pathway in hepatocytes

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At the molecular level, this dysfunction mediates pathology via several interconnected pathways

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When glutamate binds to its corresponding receptors (mainly NMDA, AMPA, and kainate receptors), sodium and calcium ions enter the postsynaptic neuron causing depolarization and therefore increasing the likelihood of an action potential, thus pressing the accelerator on the brain [8,9]