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In a study of 18 people with AD, exenatide treatment for 18 months was associated with lower plasma-sourced, neuron-derived extracellular vesicle (NDEV) A42 levels, although all other fluid biomarkers remained unchanged (62)

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Researchers evaluated medical records from more than 427,000 patients across 70 health systems

These results suggest that the inhibition of glial reaction to damage and further inflammatory processes could be considered as a promising therapy to reduce neuronal damage during PD [10]