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This review has delineated the anatomical and pathophysiological basis of rotator cuff-associated muscle atrophy, with a particular focus on the pivotal role of ROS in mediating oxidative damage, mitochondrial dysfunction, myofiber apoptosis, and activation of key catabolic signaling cascades, including the FOXO, NF-B, and MAPK pathways

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Footnotes 1.^ 2.^ REFERENCES 1 AndersonM

the level of ROS can be dramatically increased by external stimuli or internal stresses and can be toxic to cells [26]

En agissant sur la tyrosinase, en orientant la production vers une mlanine plus claire et en rduisant le stress oxydatif, il donne un teint plus lumineux, plus homogne et parfois lgrement plus clair

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