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G6PD-deficient red blood cells are highly vulnerable to oxidative damage (discussed later in this chapter), even though G6PD deficiency is never complete in humans

However, other research has suggested poorer outcomes among patients with early or specific irAEs 5

The GO-PVP-NPs affected the mitochondrial enzymatic activities, proliferation of the cells which may dominantly be attributed to the generation of ROSs

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The anti-atherosclerotic effect of Paeonol against the lipid accumulation in macrophage-derived foam cells by inhibiting ferroptosis via the SIRT1/NRF2/GPX4 signaling pathway