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Non-TNF- blocking agents are more of a mixed bag, exploiting different targets, such as antigen presenting cells (abatacept) [83], proinflammatory cytokines, and B-cell-depleting monoclonal antibodies binding CD20 (rituximab) [84], and therefore may have very different risk for the various microbes compared with the monoclonal TNF- inhibitors

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ER stress-mediated signaling: action potential and ca(2+) as key players

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The increased concentration of LBPs reached its peak after 24 h of administration, primarily identified in the liver

Pompanin, G