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A recent study also demonstrated mislocalization of APE1 from the nucleus to the cytoplasm as a possible trigger of oxidative DNA damage in spinal motor neurons expressing mutant SOD1 G93A , despite upregulation of multiple DNA repair enzymes, suggesting that restricted localization of APE1 could inhibit redox homeostasis (Li J

Moreover, DSIP demonstrates high patient tolerability and is associated with minimal side effects

Identification of selective inhibitors of uncharacterized enzymes by high-throughput screening with fluorescent activity-based probes

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Patients were allocated to three groups according to the treatment modality

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