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The proliferation rate was significantly lower in animals receiving the Gln- (6.2 1.1, p = 0.010) or the GSH-supplemented diet (4.9 1.6, p = 0.002) compared to infected animals receiving the non-supplemented diet

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This author has long recognized the need for a shift of clinical focus from glycemic status to insulin homeostasis for detecting and optimally managing adverse metabolic, proinflammatory, endothelial, developmental, and neurologic effects of hyperinsulinism (ref