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Notably, Through a feedback mechanism, mTORC1 inhibition may momentarily increase mTORC2 signaling, causing temporary metabolic disruptions during the first phase of treatment (224), which can transiently disrupt metabolism and frequently leads to hyperlipidemia, necessitating concomitant lipid-lowering therapy (237239)

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The version with DAC (Drug Affinity Complex) carries a chemical linker that binds to albumin in the blood, dramatically extending its half-life to several days

PubMed Zhang, L., Chopp, M., Lu, M., Zhang, T., Winter, S., Doppler, E., Meier, D., Chao, L., Eapen, A., Pabla, P., & Zhang, Z.G