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A nutrient-dense diet supplying methylation cofactors (folate, betaine, vitamin B12, vitamin B2, etc.) is foundational

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De novo CNV analysis implicates specific abnormalities of postsynaptic signalling complexes in the pathogenesis of schizophrenia

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This functional RBC storage lesion correlated with significant defects in glutathione (a central molecular player in cellular redox capacity), increased purine oxidation, and increased glycolysis (an expected shift in G6PD deficiency)