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As a critical molecule in cellular energy metabolism and DNA repair, insufficient NAD+ directly triggers a series of metabolic abnormalities, including impaired mitochondrial function, reduced energy metabolism efficiency, and weakened DNA repair capacity

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[7] A synthetic peptide containing the LKKTETQ domain promoted dermal wound repair in diabetic (db/db) and aged mice, which is real and interesting but it is a mouse-skin result

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Moreover, recent data suggest that oxidative injury may play a significant role in demyelination and pathogenesis in MS [116]

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