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Lipid overload meets S-palmitoylation: a metabolic signalling nexus driving cardiovascular and heart disease

For example, in conditions such as inborn errors of metabolism (e.g., medium-chain acyl-CoA dehydrogenase deficiency), accumulating acyl-CoA intermediates are converted into acylcarnitines and excreted via urine

CDDO-Methyl treatment did not impact body weight, liver damage, or serum glucose and cholesterol levels (Fig