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Arashiki N et al (2016) ATP11C is a major flippase in human erythrocytes and its defect causes congenital hemolytic anemia

Ca 2 signatures, decoded by CaMs, CDPKs, and CBLs, emerge as key regulators of metabolic plasticity, linking nutrient sensing with hormonal and transcriptional responses

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Genetics and pathogenesis of inflammatory bowel disease

In conclusion our studies indicate that oral L-GSH supplementation in individuals with T2DM for three months was able to maintain the levels of GSH, reduce oxidative stress, and diminish mycobacterial burden within in vitro generated granulomas of diabetics