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Glycolytic oxidation is the primary source of brain metabolism, and abnormal glucose metabolism is the underlying mechanism of ischemiainduced brain injury

The thyroglobulin gene on 8q24.2 codes for a large dimeric glycoprotein of 330 kd for each dimer [14, 15]

The reality is that foundational therapies, while essential, sometimes cannot fully address the deep-seated mechanisms driving the autoimmune process

The reduction of TH activity could be secondary to the degeneration of dopaminergic neurons in PD

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