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In one mouse model of sepsis-associated encephalopathy, disease onset is initiated by intestinal barrier disruption, which results in sepsis and neurological dysfunction [237]
After an initial loading phase, some patients maintain with monthly injections
2007;44:83-113.[DOI] 57
Researchers are mapping the specific hypothalamic and brainstem circuits involved in its appetite-suppressing effects and how these interact with other central metabolic regulators
European Polymer Journal Research by Arn Mignon et al., published in European Polymer Journal
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