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Inhibition of neuronal ferroptosis protects hemorrhagic brain

It has been shown that activating GLP-1 receptors on beta-cells in the pancreas enhances insulin vesicle release by a mechanism that involves the closure of K + channels, which leads to the depolarization of the cell membrane and opens voltage-dependent calcium channels (VDCC)

Conditions containing heart, coronoary, cadrio*, myo* that did not mention diabetes were considered for this analysis

Application of GABA suppresses both the spontaneous and light-evoked activity of all GCs and the GABA A R antagonist bicuculline potentiates the spontaneous and light-evoked activity of ON-type GCs [79], which suggests that GABA directly inhibits GCs

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