Boerjan W, Ralph J, Baucher M
This paper presents a comprehensive review of the pathogenic mechanism of neurological diseases, the protective mechanism of Se, and the neurological protective function of selenoproteins
This is particularly relevant because in real-world scenarios, individuals are frequently exposed to both inflammatory insults (e.g., subclinical infections) and dietary chemical agents (e.g., MSG), which may act together to worsen testicular damage via overlapping or interacting mechanisms, primarily oxidative stress and endocrine disruption
Gallic acid: a dietary polyphenol that exhibits anti-neoplastic activities by modulating multiple oncogenic targets
Akkemik E, enturk M, zgeri FB, Taer, P, iftci M (2011) In vitro effects of some drugs on human erythrocyte glutathione reductase
Upon release, Nrf2 translocates to the nucleus and binds to the antioxidant response element (ARE) located in the promoter of genes coding for antioxidant/detoxifying enzymes and scavengers
