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One such factor, forkhead box protein O1 (FoxO1), has been shown to downregulate the expression of miR-148a in hepatocytes in response to ethanol.22 This suppression of miR-148a leads to overexpression of thioredoxin-interacting protein (TXNIP), which activates the NOD-like receptor family pyrin domain-containing 3 (NLRP3) inflammasome and promotes hepatocyte pyroptosis, a form of programmed cell death associated with inflammation.22 Interestingly, restoring miR-148a levels in hepatocytes has been shown to reverse ethanol-induced steatosis and inflammation.22 This expanding body of research highlights the intricate interplay between alcohol metabolism, oxidative stress and inflammatory pathways in the liver

APAP feeding resulted in a significant decrease in total glutathione levels relative to vehicle control, with greater than a 70% reduction in total GSH levels after 3 days of exposure (116.42 5.55 uM GSH for vehicle control vs 33.67 2.35 for APAP treated)

Immunol., 92:397- 403 (1993)

11 Histologically, PUVA lentigines show a variety of features

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