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Rather than stimulating hormone release, BPC-157 appears to work primarily through nitric oxide pathways, angiogenesis, and local tissue repair mechanisms

Obesity 21 , 935943 (2013)

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Mechanistic pathways identified in animal and in vitro work include: FAK-paxillin pathway activation: BPC-157 stimulates focal adhesion kinase (FAK) and its downstream effectors, promoting fibroblast migration and tendon cell outgrowth