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Natural killer (NK) cells can trigger keratinocyte apoptosis through interaction of CD94/NKG2C with HLA-E on keratinocytes and are likely a source of the apoptotic mediator granulysin [29]

However, their combined effect on insulin levels and gastrointestinal function requires careful evaluation to avoid compounding effects like nausea or hypoglycemia

268 Inflammatory cytokines (e.g., IL-6 and TNF-) upregulate the expression of CYP24A1 in colon cancer cells by activating the NF-B pathway, which in turn leads to abnormal activation of the Wnt/-catenin pathway and promotes disease progression

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