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Unlike drugs that target a single pathway, GHK-Cu acts as a bioregulatory molecule, supporting a wide range of cellular processes

Aydin O, Ellidag HY, Eren E, Kurtulus F, Yaman A, Yilmaz N

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Recent studies have implicated PRMT1 in regulating fatty acid metabolism, suggesting that it might be a pivotal node in the complex interplay between metabolic reprogramming and renal injury [17,18,19]

These combinations ensure that the weight lost is primarily adipose tissue, preserving the muscle mass necessary for long-term metabolic health