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4 Specific role of mitochondrial energy metabolic reprogramming in AKI During AKI, metabolic reprogramming in TECs may temporarily compensate for deficits in energy supply, support cellular survival, and partially mitigate tissue damage, thereby facilitating early repair processes

doi: 10.1136/emj.7.3.148

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10.1038/nature01511 8 AgarwalA.RaiS

Epithalon also appears to inhibit the activity of Caspase-3, a key enzyme that is considered to promote apoptosis (programmed cell death)