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470 In animal models of Apelin-13-caused cardiomyocyte hypertrophy and heart failure, NCOA4-activated ferritinophagy leads to the upregulation of SFXN1 expression, causing mitochondrial iron overload and mitochondrial ROS overproduction, increased lipid peroxidation, and ultimately, ferroptosis

Random-effects models were used to calculate weighted mean differences (WMDs) or hazard ratios (HRs)

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The data are correlative, and therefore no cause-and-effect determinations can be made

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Since B12 is also essential for fetal development, particularly the development of the neural tube, low B12 can increase the risk of birth defects if left unaddressed in pregnant women