Conclusion: Our data revealed the underlying mechanism by which Semaglutide improves hepatic steatosis in T2DM+NAFLD, and might shed new light on the pathological role of miR-5120/ABHD6 in the pathogenesis of T2DM+NAFLD
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Until dedicated trials are available, the use of GLP-1RAs in AITD should be viewed as an intriguing but unproven strategy, one that underscores the evolving interface between metabolism, immunity, and endocrine care
This phenomenon might be noticeable in the first few weeks of treatment or after dose increases, though this mechanism has not been specifically studied in GLP-1 clinical trials
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"There is no standardization of the dosing, no standardization of safetyand there have been FDA reports of people ending up in the emergency room because of it." Patients should know exactly what they are being prescribed and whether it is FDA-approved
