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In humans, AA and linoleic acid are the most abundant PUFA that serve as the preferred oxidation site for LOXs using molecular oxygen to form hydroperoxyl radicals at different carbon position of acyl chains

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To estimate how the peptide levels during the first 12 months were associated with the fasting C-peptide/glucose ratio at 24 months, a linear slope for each peptide and each individual over the first four visits was calculated using linear regression models

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Then the activated PKC- triggers the release of ZO-1 and actin filament, which results in the loose conformation of TJs, increased barrier permeability and excessive abnormal antigenic transportation [84, 183]