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Mutations in the tetramer interface of human glucose6phosphate dehydrogenase reveals kinetic differences between oligomeric states

Research shows this mechanism maintains physiological GH release patterns and avoids supraphysiological spikes associated with direct growth hormone administration

This intracrine pathway operates independently of PTH and is instead driven by local inflammatory mediators such as interferon- (IFN-) and lipopolysaccharide (LPS) [96, 99]

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