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A decrease in IGF-I levels in AD and VD has been widely documented and it may be involved in the development of neurofibrillary tangles, abnormal amyloid metabolism and aberrant Tau phosphorylation, cognitive loss, neural inflammation, oxidative stress or mitochondrial dysfunction, among others[68, 325]

GIP regulates inflammation and body weight by restraining myeloid-cell-derived S100A8/A9

Limited coverage for obesity indications versus diabetes, step-therapy protocols requiring GLP-1 monotherapy trials, and prior authorization requirements could be a major limitation for market entry in early commercialization

He J, Deng C, Krall L, Shan Z

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