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2011 in the Journal of Applied Physiology characterized BPC-157's effects on tendon fibroblast outgrowth through increased FAK phosphorylation and downstream paxillin activation, with measurable effects on cell migration rate and tendon-derived fibroblast survival after mechanical injury

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Furthermore, in mouse models with defects in pericyte generation, there is a failure to downregulate genes associated with increased endothelial permeability such as angiopoietin (Angpt2) and plasmalemma vesicle-associated protein (Plvap) gene [35, 40]