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Specifically, in hypoxic polymorphonuclear MDSCs, upregulation of the fatty acid transporter FATP2 promotes the uptake of peroxidation-sensitive arachidonic acid (AA), whereas in neutrophils with decreased expression of the lysophospholipid acyltransferase MBOAT1, the accumulation of PUFA-containing phosphatidylethanolamine causes this metabolic vulnerability

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Figure 2 2.3.2 Neutrophils BAs suppress neutrophil infiltration into inflammatory sites, thereby inhibiting the onset of acute gout

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Research has shown arrhythmia prevention in models of both hyperkalemia (too much potassium) and hypokalemia (too little potassium), conditions that commonly trigger dangerous heart rhythm disturbances

The cells responsible for this effect have only recently been identified