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The in vitro studies demonstrated that we designed liposome complex could penetrate faster and more specifically into AML cells, and trigger more efficiently the ROS elevation and GSH depletion thus result in AML cell apoptosis

Because glutamine is the source of -KG, glutaminase inhibitors such as BPTES interfere with -KG homeostasis and lead to inhibited proliferation in cancer cells carrying this mutant IDH1 (Seltzer et al., 2010)

Metabolic flexibility determines human NK cell functional fate in the tumor microenvironment

It appears that the SNP of the GPx1 Pro198Leu would be influenced by modulation of ROS levels and the regulation of carcinogenesis

Ionic mechanisms in glutamate-induced astrocyte swelling: role of K + influx

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