"We have seen unprecedented growth [in metabolic health]
It is the targeted modulation of the biological systems that drive metabolic decline, body composition changes, and long-term healthspan during and after the menopausal transition
At the follicle level, GHK-Cu appears to work through several parallel mechanisms that have nothing to do with blocking androgens: What GHK-Cu does not do, based on the mechanistic literature, is directly block the 5-alpha reductase enzyme the way finasteride does
Indirectly, GHK-Cu upregulates the expression of key antioxidant enzymes including superoxide dismutase, catalase, and glutathione peroxidase
GLP-1 receptor agonists are different
Exendin-4 became the chemical scaffold for exenatide, and on April 28, 2005, the FDA approved exenatide (Byetta) for type 2 diabetes the first GLP-1 receptor agonist ever marketed