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3.3.2 Role of MOTS-c in OPG/RANKL-mediated osteoclasts In a UHMWPE model-induced osteolysis mouse model, MOTS-c was found to increase the intracellular osteoprotegerin (OPG)/RANKL ratio in osteocytes, thereby inhibiting osteoclastogenesis

A special committee investigation by the relevant institutions found that Leen Kawas (first author on Benoist 2014, co-author on McCoy 2013, and at the time CEO of the biotech commercializing a Dihexa derivative) had altered images in her doctoral dissertation and at least four published research papers from the Harding laboratory between approximately 2008 and 2012

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[DOI] [PMC free article] [PubMed] [Google Scholar] 179.Rivera E.J., Goldin A., Fulmer N., Tavares R., Wands J.R., de la Monte S.M

mito GCL was then subcloned into pcDNA3.1 + (Invitrogen) and pIRES2-DsRed2 (Clontech Laboratories Inc., Palo Alto, CA, USA) with Nhe I/ Sma I or Nhe I/ Xma I restriction enzymes, respectively

One peptide that has gained significant attention is BPC-157, known for its potential to support healing, recovery, and overall tissue health