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Grottoli S, Gauna C, Tassone F, Aimaretti G, Corneli G, Wu Z, Strasburger CJ, Dieguez C, Casanueva FF, Ghigo E, Maccario M

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Collectively, these observations militate against the idea that AA-induced alpha cell proliferation accounts for the high circulating levels of glucagon observed acutely upon administration of GRAs, an effect which we instead attribute to the removal of GLP-1(936)s suppressor effect

Winlove, P.G

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