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Extended exposure to temperatures above 77 degrees Fahrenheit accelerates degradation

Rather than directly impairing osteoclasts, it targets the signaling protein RANK ligand, which is required for osteoclast generation, thereby reducing osteoclast formation and activity through an immune-mediated mechanism

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This GABA-mediated inhibition of T cell responses may provide the mechanism of GABA-associated protection of animal models for development of T1D [134]