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By inhibiting drug-metabolizing enzymes (e.g., cytochrome P450, UDP-glucuronosyltransferase) and enhancing intestinal absorption (21, 32), PPR significantly inhibits P-glycoprotein (P-gp) and enhances the efficacy of co-administered compounds like CUR and RSV, positioning it as a critical adjunct in nutraceutical formulations (33, 34)

Moreover, compared with animal studies, the pharmacological data and rigorous clinical trial evidence of Th17/Treg regulation in human AR patients are significantly insufficient

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HCPCS J-codes typically define a specific unit of measurement (such as per 1mg or per 0.1mg)