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The 2nd glutathione-interaction site appears to preferentially interact with the -glutamylcysteinyl moiety of GSH and is thought to facilitate the recruitment and activation of GSH as a preferred reducing agent 7,11,13,17,50

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It also lowers the levels of inflammatory markers like high-sensitivity C-reactive protein (hs-CRP), thereby diminishing the intensity of the systemic inflammatory response

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130 Due to the absence of the uricase enzyme responsible for converting uric acid into allantoin and allantoic acid, UA remains the terminal metabolic product in humans