Additionally, leveraging NRF2 activation can bolster cellular antioxidant defenses, further mitigating the pathological effects of ferroptosis
By controlling T cell metabolic pathways like glycolysis, OXPHOS, FAO, and amino acid metabolism, these tactics may effectively regulate T cell differentiation, function, and immune response, offering a fresh approach to the treatment of autoimmune diseases ( Figure 3 )
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Notably, aging-related AE1 protein conformational changes result in the appearance of a senescent-specific antigen recognized by autologous antibodies [75]
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17 GSTs catalyze glutathionylation or S-glutathionylation, a reversible posttranslational modification regulating protein function, which is significantly promoted by reactive nitrogen species and reactive oxygen species (ROS)
