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The addition of exogenous ROS results in resensitization of cells with high VRK2 expression to GSH inhibition, which is consistent with the fact that PC cells with low expression of VRK2 are susceptible to GSH inhibition attributes to its high basal ROS level, suggesting that cells in a state of oxidative stress have enhanced dependence on the GSH system

Hence, our objectives were to (1) characterize EVs released from BUTE cells, (2) determine how estradiol and progesterone affect EV release, and (3) determine how ovarian steroids affect the metabolic content of EVs released from BUTE cells

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FIGURE 1 A systematic literature search was performed across the PubMed database for relevant studies published up to June 2025

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J Immunol (2006) 176:462231