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Collins suspected these side effects occurred when antibiotics triggered oxidative stress a condition in which cells produce chemically reactive oxygen molecules that damage the bacterias DNA and enzymes, as well as the membrane that encloses the cell

Particularly in the early stages of the disease

Combined triad-GSH metabolism model simulations: impact of quinone metabolism on glutathione homeostasis The model was used to investigate how single vs constant 50 M exposure of doxorubicin influenced blood and cytosolic GSH and cysteine (Cys) levels, over a 10-hour time-span, shown in Fig

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