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Hala M, Ebtisam E, Sanaa I, Badran M, Marwa A, Said M

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While NO is largely implicated in hemostatic mechanisms, in tail-amputation-models under heparin- and warfarin-administration, both the NO-synthase (NOS)-blocker, L-NAME (prothrombotic) and the NOS-substrate L-arginine (antithrombotic), were little investigated

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2D), a phenotype closely resembling liver PSAT1 KO mice